李佳钰,刘瑞金,于凯江. 半胱氨酸蛋白酶在脓毒症中的研究进展[J]. 中国急救医学, 2019, 39(4): 398-400.
Li Jia-yu, Liu Rui-jin, Yu Kai-jiang. Research progress of Caspase in sepsis. Chinese Journal of Critical Care Medicine, 2019, 39(4): 398-400.
[1]Lakshmikanth CL, Jacob SP, Chaithra VH, et al. Sepsis: in search of cure[J]. Inflamm Res, 2016, 65(8): 587-602.
[2]Nolan A, Weiden MD. Trends in sepsis and infection sources in the United States. A population-based study[J]. Ann Am Thorac Soc, 2015, 12(5): 784.
[3]Aziz M, Jacob A, Wang P. Revisiting caspases in sepsis[J]. Cell Death Dis, 2014, 11(5): e1526.
[4]Khalilzadeh B, Shadjou N, Kanberoglu GS, et al. Advances in nanomaterial based optical biosensing and bioimaging of apoptosis via caspase-3 activity: a review[J]. Mikrochim Acta, 2018, 185(9): 434.
[5]Wang YC, Liu QX, Liu T, et al. Caspase-1-dependent pyroptosis of peripheral blood mononuclear cells predicts the development of sepsis in severe trauma patients: A prospective observational study[J]. Medicine (Baltimore), 2018, 97(8): e9859.
[6]Weng D, Marty-Roix R, Ganesan S, et al. Caspase-8 and RIP kinases regulate bacteria-induced innate immune responses and cell death[J]. Proc Natl Acad Sci USA, 2014, 111(20): 7391-7396.
[7]Lee H, Shin EA, Lee JH, et al. Caspase inhibitors: a review of recently patented compounds (2013-2015)[J]. Expert Opin Ther Pat, 2018, 28(1): 47-59.
[8]Su H, Bidere N, Zheng L, et al. Requirement for caspase-8 in NF-kappa B activation by antigen receptor[J]. Science, 2005, 307(5714): 1465-1468.
[9]Yan B, Wei K, Hou L, et al. Receptor-Interacting Protein 3/Caspase-8 May Regulate Inflammatory Response and Promote Tissue Regeneration in the Periodontal Microenvironment[J]. Med Sci Monit, 2018, 24: 5247-5257.
[10]Sagulenko V, Lawlor KE, Vince JE. New insights into the regulation of innate immunity by caspase-8[J]. Arthritis Res Ther, 2016, 13, 18: 4.
[11]Shimizu T, Nakazato T, Xian MJ, et al. Resveratrol induces apoptosis of human malignant B cells by activation of caspase-3 and p38 MAP kinase pathways[J]. Biochem Pharmacol, 2006, 71(6): 742-750.
[12]Parlato M, Souza-Fonseca-Guimaraes F, Philippart F, et al. CD24-triggered caspase-dependent apoptosis via mitochondrial membrane depolarization and reactive oxygen species production of human neutrophils is impaired in sepsis[J]. J Immunol, 2014, 192(5): 2449-2459.
[13]Exline MC, Justiniano S, Hollyfield JL, et al. Microvesicular caspase-1 mediates lymphocyte apoptosis in sepsis[J]. PLoS One, 2014, 9(3): e90 968.
[14]Hu Z, Murakami T, Suzuki K, et al. Antimicrobial cathelicidin peptide LL-37 inhibits the LPS/ATP-induced pyroptosis of macrophagesby dual mechanism[J]. PLoS One, 2014, 9(1): e85 765.
[15]Vanden Berghe T, Demon D, Bogaert P, et al. Simultaneous targeting of IL-1 and IL-18 is required for protection against inflammatory and septicshock[J]. Am J Respir Crit Care Med, 2014, 189(3): 282-291.
[16]Aachoui Y, Sagulenko V, Miao EA, et al. Inflammasome-mediated pyroptotic and apoptotic cell death, and defense against infection[J]. Curr Opin Microbiol, 2013, 16(3): 319-326.
[17]Sarkar A, Balakrishnan K, Chen J, et al. Molecular evidence of Zn chelation of the procaspase activating compound B-PAC-1 in B cell lymphoma[J]. Oncotarget, 2016, 7(3): 3461-3476.
[18]Hagar JA, Miao EA. Detection of cytosolic bacteria by inflammatory caspases[J]. Curr Opin Microbiol, 2014, 17: 61-66.
[19]Larios Mora A, Detalle L, Gallup JM, et al. Delivery of ALX-0171 by inhalation greatly reduces respiratory syncytial virus disease in newborn lambs[J]. MAbs, 2018, 10(5): 778-795.
[20]Chen H, Fang Y, Wu J, et al. RIPK3-MLKL-mediated necroinflammation contributes to AKI progression to CKD[J]. Cell D eath Dis, 2018, 9(9): 878.
[21]Duprez L, Takahashi N, Van Hauwermeiren F, et al. RIP kinase-dependent necrosis drives lethal systemic inflammatory response syndrome[J]. Immunity, 2011, 35(6): 908-918.
[22]Hutchins NA, Unsinger J, Hotchkiss RS, et al. The new normal: immunomodulatory agents against sepsis immune suppression[J]. Trends Mol Med, 2014, 20(4): 224-233.