脓毒症心功能不全机制的研究进展

doi:10.3969/j.issn.1002-1949.2018.09.01

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摘要脓毒症心功能不全是脓毒症的严重并发症，但是其发病机制尚未明确。目前研究表明，炎症、内皮细胞损伤、氧化应激、线粒体功能障碍、心肌细胞凋亡在脓毒症心功能不全中起着重要作用。本文对脓毒症心功能不全发病机制的研究进展进行综述。

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	李亚敏
	乔建峰
	张雯瑄
	彭振宇

关键词 ：脓毒症, 心功能不全, 炎症, 氧化应激, 内皮细胞损伤, 线粒体功能障碍, 凋亡

基金资助:中南大学研究生创新课题（2017zzts880）

通讯作者: 彭振宇（1980-），男，博士，主治医师，硕士生导师，E-mail：pengzhenyu1999@csu.edu.cn。

作者简介: 李亚敏（1975-），女，主任护师，博士，硕士生导师，E-mail：amin5433@163.com。

引用本文:

李亚敏，乔建峰，张雯瑄，彭振宇. 脓毒症心功能不全机制的研究进展[J]. 中国急救医学, 2018, 38(9): 819-822.
Li Ya-min, Qiao Jian-feng, Zhang Wen-xuan, Peng Zhen-yu. The advances on the mechanisms of sepsis-induced cardiac dysfunction. Chinese Journal of Critical Care Medicine, 2018, 38(9): 819-822.

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http://www.cnemergency.org.cn/CN/10.3969/j.issn.1002-1949.2018.09.01 或 http://www.cnemergency.org.cn/CN/Y2018/V38/I9/819

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